| 1999 | | A sesquiterpene, dehydrocostus lactone, inhibits the expression of inducible nitric oxide synthase and TNF-alpha in LPS-activated macrophages. | 이수환, 이희주 |
| 2001 | | Activation of mouse macrophage by soluble endogenous murine leukemia virus (MuLV) envelope protein. | 강엽 |
| 2003 | | Amyloid beta peptide-induced corpus callosum damage and glial activation in vivo. | 김승업 |
| 2021 | | Anti-inflammatory Effects of Empagliflozin and Gemigliptin on LPS-Stimulated Macrophage via the IKK/NF- κ B, MKK7/JNK, and JAK2/STAT1 Signalling Pathways | 강엽, 김대중, 김혜진, 이관우, 이나미, 전자영, 최성이, 한승진, 허유정 |
| 2010 | | Endogenous expression of interleukin-4 regulates macrophage activation and confines cavity formation after traumatic spinal cord injury. | 김병곤 |
| 2010 | | Enhanced phosphatidylinositol 4-phosphate 5-kinase alpha expression and PI(4,5)P2 production in LPS-stimulated microglia. | 이상윤, 조은혜, 주일로 |
| 2022 | | Hepatoprotective effects of gemigliptin and empagliflozin in a murine model of diet-induced non-alcoholic fatty liver disease | 강엽, 김대중, 김혜진, 이관우, 이나미, 전자영, 최성이, 한승진, 허유정 |
| 2009 | | Identification of the amino acid sequence motif of alpha-synuclein responsible for macrophage activation. | 박상면 |
| 2005 | | Inhibition of thrombin-induced microglial activation and NADPH oxidase by minocycline protects dopaminergic neurons in the substantia nigra in vivo. | 김승업, 진병관 |
| 2018 | | Molecular mechanisms of biogenesis of apoptotic exosome-like vesicles and their roles as damage-associated molecular patterns | 김경민, 박선, 신호준, 최용준 |
| 1999 | | Prevention of encephalomyocarditis virus-induced diabetes in mice by inhibition of the tyrosine kinase signalling pathway and subsequent suppression of nitric oxide production in macrophages. | 윤지원 |
| 2001 | | Role of Hck in the pathogenesis of encephalomyocarditis virus-induced diabetes in mice. | 김현만, 최경숙 |
| 1997 | | Role of macrophages and macrophage-derived cytokines in the pathogenesis of Kilham rat virus-induced autoimmune diabetes in diabetes-resistant BioBreeding rats. | 강엽, 윤지원 |
