Genomic Instability Research Center13

Pub YearTitleAJOU
Author
-Age-dependent decline of BubR1 promotes Chromosome Mis-segregation in HDF cells-
-Age-dependent decline of BubR1 promotes Chromosome Mis-segregation in HDF cells-
-ATM-dependent chromatin remodeler Rsf-1 facilitates DNA damage checkpoints and homologous recombination repair-
-Defective Base Excision Repair resulting from DNA Polymerase β inactivation in the nervous system-
-DNMT1 is the responsible for RIP3 maintaing as silencing in cancer cell lines-
-A formulated red ginseng extract inhibits autophagic flux and sensitizes to doxorubicin-induced cell death-
-Inhibition of Cdk1 by ROS induces nuclear shape alteration possibly via formation of aggregated lamin during mitosis.-
-The novel mechanism for post-translational modification of RIP3-
-Oxidative stress during mitosis induces BAF mislocalization through decreasing PP2A activity, leading to abnormal nuclear shape-
-Remodeling and spacing factor 1 (RSF1) promotes DSB-induced transcriptional silencing by recruiting EZH2 polycomb transcriptional repressor at DSB sites-
-The role of ATM responding to DNA damage induced by Xrcc1 deficiency during oligodendrocyte genesis-
-RSF1 modifies chromatin structure in response to DNA damage to propagate DNA damage checkpoint signal-
-RSF1, chromatin remodeling protein, facilitates DNA damage checkpoints and homologous recombination repair-
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